Was Earth ever infected by martian biota? Clues from radioresistant bacteria

Here we propose that the radioresistance (tolerance to ionizing radiation) observed in several terrestrial bacteria has a martian origin. Multiple inconsistencies with the current view of radioresistance as an accidental side effect of tolerance to desiccation are discussed. Experiments carried out 25 years ago were reproduced to demonstrate that \"ordinary\" bacteria can develop high radioresistance ability after multiple cycles of exposure to high radiation dosages followed by cycles of recovery of the bacterial population. We argue that \"natural\" cycles of this kind could have taken place only on the martian surface, and we hypothesize that Mars microorganisms could have developed radioresistance in just several million years' time and, subsequently, have undergone transfer to Earth by way of martian meteorites. Our mechanism implies multiple and frequent exchanges of biota between Mars and Earth.     

激波风洞重模型气动力试验研究

在激波风洞上进行气动力试验时,风洞启动时巨大的冲击载荷使模型-天平受到充分的激励,从而形成惯性干扰力,并与真实气动力混杂在一起,甚至完全覆盖气动力,降低了试验精准度,使得试验模型的质量受到极大的限制.本文介绍了CARDC-dia.2米激波风洞进行大、重模型的压电天平气动力试验研究情况,包括天平设计、天平校准、惯性补偿和风洞试验等几个方面.研究结果表明:气动力试验模型质量可从过去的500g增加到8kg,模型长度可达1m.从而提高了激波风洞测力试验能力,能满足高超声速飞行器试验的需求.     

Spaceflight and clinorotation cause cytoskeleton and mitochondria changes and increases in apoptosis in cultured cells.

The cytoskeleton is a complex network of fibers that is sensitive to environmental factors including microgravity and altered gravitational forces. Cellular functions such as transport of cell organelles depend on cytoskeletal integrity; regulation of cytoskeletal activity plays a role in cell maintenance, cell division, and apoptosis. Here we report cytoskeletal and mitochondria alterations in cultured human lymphocyte (Jurkat) cells after exposure to spaceflight and in insect cells of Drosophila melanogaster (Schneider S-1) after exposure to conditions created by clinostat rotation. Jurkat cells were flown on the space shuttle in Biorack cassettes while Schneider S-1 cells were exposed to altered gravity forces as produced by clinostat rotation. The effects of both treatments were similar in the different cell types. Fifty percent of cells displayed effects on the microtubule network in both cell lines. Under these experimental conditions mitochondria clustering and morphological alterations of mitochondrial cristae was observed to various degrees after 4 and 48 hours of culture. Jurkat cells underwent cell divisions during exposure to spaceflight but a large number of apoptotic cells was also observed. Similar results were obtained in Schneider S-1 cells cultured under clinostat rotation. Both cell lines displayed mitochondria abnormalities and mitochondria clustering toward one side of the cells which is interpreted to be the result of microtubule disruption and failure of mitochondria transport along microtubules. The number of mitochondria was increased in cells exposed to altered gravity while cristae morphology was severely affected indicating altered mitochondria function. These results show that spaceflight as well as altered gravity produced by clinostat rotation affects microtubule and mitochondria organization and results in increases in apoptosis. Grant numbers: NAG 10-0224, NAG2-985.     

Pre-storm behaviour of NmF2 and TEC (GPS) over equatorial and low latitude stations in the Indian sector

The pre-storm behavior of NmF₂ and TEC over an equatorial station, Trivandrum (8.47°N, 76.91°E, dip 0.6°S) and a low latitude station, Waltair (17.7°N,83.3°E, dip 20°N) has been studied for a total of 18 strong geomagnetic storms with DST ? −100 nT. The simultaneous measurements of GPS-TEC and NmF₂ over Trivandrum and Waltair during the period 2000–2005 have been considered for the present study. It is found that there is a substantial increase in NmF₂ and TEC before the onset of the storm over Waltair, while the increase is not present at Trivandrum. The origin of pre-storm enhancements in electron density still remains unresolved owing to several conditions in their potential sources and occurrence mechanisms. In the present study an attempt is made to identify the possible mechanisms responsible for such enhancements in electron density of the F-region.