Space–Time Structure of Energetic Electron Precipitations according to the Data of Balloon Observations and Polar Satellite Measurements on February 1–6, 2015

Cosmic Research - The results of an analysis of the space–time characteristics and dynamics of precipitations of magnetospheric electrons with energies in the range from 0.1 to 0.7 MeV are...     

Water Partitioning in Planetary Embryos and Protoplanets with Magma Oceans

The water content of magma oceans is widely accepted as a key factor that determines whether a terrestrial planet is habitable. Water ocean mass is determined as a result not only of water delivery and loss, but also of water partitioning among several reservoirs. Here we review our current understanding of water partitioning among the atmosphere, magma ocean, and solid mantle of accreting planetary embryos and protoplanets just after giant collisions. Magma oceans are readily formed in planetary embryos and protoplanets in their accretion phase. Significant amounts of water are partitioned into magma oceans, provided the planetary building blocks are water-rich enough. Particularly important but still quite uncertain issues are how much water the planetary building blocks contain initially and how water goes out of the solidifying mantle and is finally degassed to the atmosphere. Constraints from both solar-system explorations and exoplanet observations and also from laboratory experiments are needed to resolve these issues.     

Advanced Pulsed Plasma Thrusters and Their Application as a Part of Small Spacecraft Propulsion Systems

The paper presents the research results of the effect of a capacitor energy storage device configuration on the specific characteristics of advanced modern propulsion systems based on the ablative pulsed plasma thrusters (APPT). These thrusters are designed to perform specific tasks within the small spacecrafts with the onboard power capacity up to 200 W.     

Role of Equatorial Anomaly in Earthquake time precursive features: A few strong events over West Pacific zone

The earthquake (EQ) time coupling processes between equator-low-mid latitude ionosphere are complex due to inherent dynamical status of each latitudinal zone and qualified geomagnetic roles working in the system. In an attempt to identify such process, the paper presents temporal and latitudinal variations of ionization density (foF₂) covering 45°N to 35°S, during a number of earthquake events (M?>?5.5). The approaches adopted for extraction of features by the earthquake induced preparatory processes are discussed in the paper through identification of parameters like the ‘EQ time modification in density gradient’ defined by δ?=?(foF_{2 max}???foF_{2 min})∕τ_mm, where τ_mm – time span (in days) between EQ modified density maximum and minimum, and the Earthquake time Equatorial Anomaly, i.e. EEA, one of the most significant phenomenon which develops even during night time irrespective of epicenter position. Based on the observations, the paper presents the seismic time coupling dynamics through anomaly like manifestations between equator, low and mid latitude ionosphere bringing in the global Total Electron Content (TEC) features as supporting indices.     

Optical Design for a Cubesat: Unobscured Telescope,Using Freeform Mirrors and a Curved Detector

The possibility of constructing an optical scheme of a telescope for a small satellite based on mirrors with freeform surfaces and a curved photodetector is considered. It is shown that the use of a new element base allows the effective area of the main mirror to be maximized and the field of view up to 9.4 times to be increased in comparison with the classical scheme of the Ritchey–Chretien scheme in ensuring the high image quality.     

Influence of the 2:1 resonance in the orbits of GPS satellites

Due to the characteristics of their orbits the GPS satellites are submitted to the following main perturbations: terrestrial gravitational field, luni-solar gravitational attraction and solar radiation pressure (including the effects of the Earth's shadow). An additional perturbation arises due to the 2:1 commensurability of the orbital period of the satellite with the period of the Earth's rotation. An analytical theory is briefly presented to solve the equations of motion including the previously mentioned effects. The analytical solution, based on the Lie-Hori method, is compared with a numerical integration of the equations.     

Gravity effects on connective tissue biosynthesis by cultured mesenchymal cells.

Quantitative and qualitative aspects of collagen synthesis under microgravity, normal gravity and hypergravity conditions were investigated during the spacelab D-2 mission by incubating human fibroblast cultures with [3H]-proline for 0, 4, 7, 10 and 20 hours. Quantitative analysis revealed an increase of collagen synthesis under microgravity conditions, being 40% higher than 1g controls. Hypergravity samples at 1.44g, 6.6g and 10g showed a decrease in collagen synthesis with increasing g, being down to about 15% at 10g. The relative proportion of collagen from total protein synthesized, the secretion of collagen by the cells, proline hydroxylation of individual collagen alpha-chains and the relative proportions of collagens I, III and V synthesized were not affected at any of the applied conditions.     

DNA damage and repair in oncogenic transformation by heavy ion radiation.

Energetic heavy ions are present in galactic cosmic rays and solar particle events. One of the most important late effects in risk assessment is carcinogenesis. We have studied the carcinogenic effects of heavy ions at the cellular and molecular levels and have obtained quantitative data on dose-response curves and on the repair of oncogenic lesions for heavy particles with various charges and energies. Studies with repair inhibitors and restriction endonucleases indicated that for oncogenic transformation DNA is the primary target. Results from heavy ion experiments showed that the cross section increased with LET and reached a maximum value of about 0.02 micrometer2 at about 500 keV/micrometer. This limited size of cross section suggests that only a fraction of cellular genomic DNA is important in radiogenic transformation. Free radical scavengers, such as DMSO, do not give any effect on induction of oncogenic transformation by 600 MeV/u iron particles, suggesting most oncogenic damage induced by high-LET heavy ions is through direct action. Repair studies with stationary phase cells showed that the amount of reparable oncogenic lesions decreased with an increase of LET and that heavy ions with LET greater than 200 keV/micrometer produced only irreparable oncogenic damage. An enhancement effect for oncogenic transformation was observed in cells irradiated by low-dose-rate argon ions (400 MeV/u; 120 keV/micrometer). Chromosomal aberrations, such as translocation and deletion, but not sister chromatid exchange, are essential for heavy-ion-induced oncogenic transformation. The basic mechanism(s) of misrepair of DNA damage, which form oncogenic lesions, is unknown.     

The role of DNA repair on cell killing by charged particles.

It can be noted that it is not simple double strand breaks (dsb) but the non-reparable breaks that are associated with high biological effectiveness in the cell killing effect for high LET radiation. Here, we have examined the effectiveness of fast neutrons and low (initial energy = 12 MeV/u) or high (135 MeV/u) energy charged particles on cell death in 19 mammalian cell lines including radiosensitive mutants. Some of the radiosensitive lines were deficient in DNA dsb repair such as LX830, M10, V3, and L5178Y-S cells and showed lower values of relative biological effectiveness (RBE) for fast neutrons if compared with their parent cell lines. The other lines of human ataxia-telangiectasia fibroblasts, irs 1, irs 2, irs 3 and irs1SF cells, which were also radiosensitive but known as proficient in dsb repair, showed moderated RBEs. Dsb repair deficient mutants showed low RBE values for heavy ions. These experimental findings suggest that the DNA repair system does not play a major role against the attack of high linear energy transfer (LET) radiations. Therefore, we hypothesize that a main cause of cell death induced by high LET radiations is due to non-reparable dsb, which are produced at a higher rate compared to low LET radiations.